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never get the disease, hinting at an unknown environmental trigger could play a role in the development of this chronic autoimmune condition., a new study led by researchers from Cardiff University in the UK points in a different direction: Type 1 diabetes might start with proteins on bacteria, sparking an ominous shift in the immune system.
Insulin helps glucose move from the bloodstream into our cells, which use it for energy. It's a vital hormone produced by beta cells in the pancreas, and without it, the body's blood sugar can surge to dangerously high levels., Sewell and his colleagues linked the loss of insulin-producing tissues with killer T cells – a class of white blood cells that kill certain other cells, includingcells or cells infected by a pathogen.
The team conducted lab experiments to simulate such infections, introducing bacterial proteins to cell lines from non-diabetic human donors and observing how the donors' killer T cells reacted. By demystifying the origins of type 1 diabetes, the researchers say, we may reveal new ways to treat the disease – or maybe even learn how to prevent it.
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