Tirzepatide Reduces Circulatory Overload and End-Organ Damage in HFpEF and Obesity

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Tirzepatide Reduces Circulatory Overload and End-Organ Damage in HFpEF and Obesity
TirzepatideHeart FailurePreserved Ejection Fraction
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A secondary analysis of the SUMMIT trial indicates that tirzepatide, when used in patients with obesity-related heart failure with preserved ejection fraction (HFpEF), decreases blood pressure and blood volume. It also improves cardiovascular function and reduces systemic inflammation.

In a secondary analysis of the SUMMIT trial, patients with obesity-related heart failure with preserved ejection fraction (HFpEF) who were treated with tirzepatide showed reduced systolic blood pressure and decreased blood volume. The treatment also correlated with lower levels of microalbuminuria, improved cardiovascular quality of life, and increased walking distance. Additionally, reduced C-reactive protein levels were associated with lower troponin T levels and improved walking distance.

These findings suggest that tirzepatide reduces circulatory volume-pressure overload, systemic inflammation, and cardiovascular-kidney end-organ injury

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Tirzepatide Heart Failure Preserved Ejection Fraction Obesity SUMMIT Trial Circulatory Overload

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