The exciting achievement came about after researchers were successfully able to interfere with an enyzme typically found to be overactive in the brains of those suffering from the disease.
Early tests conducted on mice revealed significant — and promising — results.
“We saw wonderful effects in terms of reducing neurodegeneration and neuroinflammatory responses, and even rescuing behavior deficits.”— particularly dementia brought on by CDK5 overactivity. The peptide is also showing strong results in mending the brain’s tau protein, which when altered, becomes a main characteristic of AD.
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