Al Horford: “I personally worry [about] and not only career-wise, but just the rest of your life.” Kawhi Leonard: “If you do get it, will it affect you to be able to play again? Everyone knows it hits everybody’s body different.”
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In the lungs, “the best available data suggests that it’s probably no more than 5% of individuals with COVID will go on to develop scarring or a fibrotic condition,” said James Hull, a sports respiratory specialist in London who has worked with the Institute of Sport, Exercise and Health. “But I’m talking about people who’ve been hospitalized. The signal of people getting long-term fibrotic lung manifestations, and who have not been hospitalized, is going to be very, very low.
Christopher Cooper, a professor emeritus of medicine and physiology at UCLA’s David Geffen School of Medicine who has led the exercise physiology research lab, called it important to seek scientific evidence without being prematurely alarmist.“People with asymptomatic infection or mild disease are not likely to be at risk” of decreased heart or lung function, Cooper said.
With athletes being tested before they return to fields and courts, better data targeting COVID-19’s effects on them could emerge in the next few months, and if leagues share that data, researchers could draw stronger conclusions. The NBA and its players association supported a league-wide study in the spring, led by the Mayo Clinic, that sought to understand what percentage of the league’s employees and players had COVID-19 antibodies.
“I’m looking after an international triathlete who is at the very peak of their game to having a cough, a loss of smell — classical, clinical symptoms,” Hull said. “And here we are, four months out and they’re crippled by their symptoms, and those symptoms are nebulous. Fatigue, chest pain, chest discomfort, squeezing sensation, an intermittent cough, and yet blood markers and [electrocardiograms] don’t reveal a pathological explanation for it.
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