New research findings may contribute to the understanding of pathophysiology and help validate novel therapies for the prevention and treatment of COVID-19. SARS-CoV-2 infection can cause lasting damage to sensory nerves, leading to symptoms of Long COVID, according to a study by researchers from
A study conducted by researchers from Boston University, Icahn School of Medicine at Mount Sinai, and New York University has revealed that SARS-CoV-2 infection can lead to lasting damage to the dorsal root ganglia, resulting in symptoms of Post-COVID Conditions or Long COVID. The study found that thousands of genes associated with neurodegeneration and pain-related pathways were affected even after the viral infection had been cleared.
COVID-19, the disease resulting from SARS-CoV-2 infection, is associated with highly variable clinical outcomes that range from asymptomatic disease to death. For those with milder infections, COVID-19 can produce respiratory infection symptoms and sensory phenotypes such as headache and loss of sense of smell. In more severe cases, SARS-CoV-2 infection can affect nearly every organ and result in strokes from vascular occlusion, cardiovascular damage and acute renal failure.
“Several studies have found that a high proportion of Long COVID patients suffer from abnormal perception of touch, pressure, temperature, pain or tingling throughout the body.
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