Rheumatoid arthritis affects 1 in 100 people worldwide.
The origins of the antibodies that cause rheumatoid arthritis have been an area of study for many years. Somethat these antibodies can start forming at sites like the mouth, lung and intestines over 10 years before symptoms arise. But until now, it was unclear why researchers were finding these antibodies in these particular areas.
Since microbes commonly live at the same sites as the antibodies driving rheumatoid arthritis, we hypothesized that these bacteria could be triggering the production of these antibodies. We reasoned that though these antibodies were meant to attack the bacteria, rheumatoid arthritis develops when they spread beyond the intestines to attack the joints.
We found that one previously unknown species of bacteria was present in the intestines of around 20 percent of people who were either diagnosed with rheumatoid arthritis or produce the antibodies that cause the disease.Subdoligranulum didolesgii We also found that these bacteria can activate specialized immune cells called T cells in people with rheumatoid arthritis. T cells drive inflammatory responses in the body, and have been linked to the development ofThese findings suggest that these gut bacteria may be activating the immune systems of people with rheumatoid arthritis. But instead of attacking the bacteria, their immune system attacks the joints.
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