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Recent research identifies a specific fatty acid metabolite in neonatal blood that influences the severity of autism spectrum disorder symptoms. This discovery could enable early diagnosis and intervention, potentially improving outcomes for those affected by ASD.
Although the exact causes of ASD are unclear, currently available evidence points to neuroinflammation as a major factor. Several studies in mouse models of ASD have hinted at the importance of polyunsaturated fatty acids and their metabolites during pregnancy in playing a key role in ASD development. PUFA metabolites regulated by the cytochrome P450 affect fetal development in mice causing impairments closely linked to ASD symptoms.
After careful statistical analyses of the results, the researchers identified one compound in cord blood that may have strong implications for ASD severity, namely 11,12- dihydroxyeicosatrienoic acids , a dihydroxy fatty“The levels of diHETrE, an arachidonic acid-derived diol, in cord blood at birth significantly impacted subsequent ASD symptoms in children and were also associated with impaired adaptive functioning.
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