Emily is a health news writer based in London, United Kingdom. She holds a bachelor's degree in biology from Durham University and a master's degree in clinical and therapeutic neuroscience from Oxford University. She has worked in science communication, medical writing and as a local news reporter while undertaking journalism training.
Defective mitochondria may disrupt the gut microbiome, driving the development of Crohn's disease, new research in mice suggests.
Research in patients suggests that Crohn's may be partly caused by changes in the composition and function of the gut microbiome, the collection of microbes that live inside the digestive tract. Those microbes can influence inflammatory cells of the immune system, so when they change, the immune system changes in turn.Now, a study published Aug. 14 in the journal Cell Host & Microbe may explain why these microbial changes occur.
By submitting your information you agree to the Terms & Conditions and Privacy Policy and are aged 16 or over.When the team took tissue samples from the intestines of the mice and analyzed them under the microscope, they found signs of inflammation and tissue injury that resembled Crohn's disease in humans. For instance, they saw a decline in the amount of mucus made by the intestinal tissue, which would normally protect the organ's inner lining.
Bacteroides normally live in the guts of mammals, including humans, without causing a stir. However, they can become opportunistic pathogens, meaning that they may seize the opportunity to cause infections if, for instance, the intestinal wall is compromised.
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