Long COVID: Could mono virus or fat cells be playing roles?

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Long COVID: Could mono virus or fat cells be playing roles?
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Plagued by eerily similar symptoms, a British historian, an Italian archaeologist and an American preschool teacher who have never met in person are credited with describing, naming and helping bring long COVID into the public’s consciousness.

support group from the trucker cap she’d been wearing, and “long hauler” soon became part of the pandemic lexicon.

Women’s bodies also tend to have more fat tissue and emerging research suggests the coronavirus may hide in fat after infection. Scientists also are studying whether women’s fluctuating hormone levels may increase the risks. Pope, 46, started chronicling what she was experiencing in March 2020: flu-like symptoms, then her lungs, heart and joints were affected. After a month she started having some “OK” days, but symptoms persisted.

Epstein-Barr virus is among the most common of these herpes viruses: An estimated 90% of the U.S. population has been infected with it. The virus can cause mononucleosis or symptoms that may be dismissed as a cold. Stanford University researchers are among those who have found evidence that the coronavirus can infect fat cells. In a recent study, they found the virus and signs of inflammation in fat tissue taken from people who had died from COVID.

They plan to study whether injections of a manufactured antibody could reduce leptin levels — and in turn inflammation from coronavirus infections or long COVID.

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