Findings show people with long COVID-19 respond differently to COVID-19 vaccines. A new study by investigators from the Smidt Heart Institute at Cedars-Sinai suggests long COVID-19 might be caused by a dysfunction of the immune system. The study, published in the journal BMC Infectious Disease
A study from Cedars-Sinai’s Smidt Heart Institute suggests that immune system dysfunction could be causing long COVID-19. The study found that patients with long COVID-19 produced antibodies against the virus for an extended period after vaccination, with especially high levels of nucleocapsid antibodies. The implications of this sustained immune response are still unclear, and researchers are now seeking a definitive biomarker for diagnosing and understanding long COVID-19.
“We examined one part of the immune system response, the production of antibodies, which is mediated by immune cells called B-cells,” Le explained. “What you would expect after getting a COVID-19 vaccination is a jump in your spike protein antibody levels, but you wouldn’t expect a significant increase in nucleocapsid antibody levels,” said Susan Cheng, MD, MPH, the Erika J. Glazer Chair in Women’s Cardiovascular Health and Population Science, director of the Institute for Research on Healthy Aging in the Department of Cardiology at the Smidt Heart Institute, and a senior author of the study.
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