Does a family history of obesity make people more susceptible to weight gain? And if so, can we fight genetics? Well, yes and no.
The Western diet has primarily been credited with the rise in obesity along with environmental changes, not necessarily due to human genetics. However, studies show aComparing Genes Against Environmental Factors
of the fat mass– and obesity-associated gene in an individual predisposes that person to weigh 3 kg more than people who don't carry copies ofrisk allele feel hungrier and have higher levels of the hunger hormone ghrelin after a meal than those without it. The single gene mutation and syndromic forms of obesity where the environmental contribution is limited are conducive to treatment because the mutation is single and easily understood, and the antidote is available, such as recombinant leptin for the single gene mutation in thegene, where patients are leptin deficient and exhibit extreme insatiable hunger due to inactivation of the proopiomelanocortin neuron system.
The bottom line regarding this PGS score is that it's useless in predicting those at risk for obesity based on genes, due to the influence of external factors, some of which are still little understood.
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