Science, Space and Technology News 2023
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The findings suggest that an interplay of genetic and epigenetic changes feed on each other to drive the pathological manifestations of the disease. In the study led by Mathys, the researchers also compared gene expression patterns in people who showed cognitive impairments and those who did not, including some who remained sharp despite having some degree of amyloid buildup in the brain, a phenomenon known as cognitive resilience. That analysis revealed that cognitively resilient people had larger populations of two subsets of inhibitory neurons in the prefrontal cortex.
This study builds on a 2015 paper from Tsai and Kellis in which they found that many of the genome-wide association study variants associated with Alzheimer’s disease are predominantly active in immune cells like microglia, much more than in neurons or other types of brain cells.
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