Researchers have discovered that the transporter protein Mfsd2a is critical for regulating brain cells that maintain myelin sheaths, which protect nerves. This discovery could help reduce the effects of aging on the brain and lead to therapies for neurological disorders stemming from reduced myelina
Scientists from Singapore have demonstrated the critical role played by a special transporter protein in regulating the brain cells that ensure nerves are protected by coverings called myelin sheaths. The findings, reported by researchers at Duke-NUS Medical School and the National University of Singapore in the, could help to reduce the damaging impacts of aging on the brain.
“Loss of myelin sheaths occurs during the normal aging process and in neurological diseases, such as multiple sclerosis anddisease,” said Dr. Sengottuvel Vetrivel, Senior Research Fellow with Duke-NUS’ Cardiovascular & Metabolic Disorders Program and lead investigator of the study. “Developing therapies to improve myelination—the formation of the myelin sheath—in aging and disease is of great importance to ease any difficulties caused by declining myelination.
“Our study indicates that LPC omega-3 lipids act as factors within the brain to direct oligodendrocyte development, a process that is critical for brain myelination,” explained Professor David Silver, the senior author of the study and Deputy Director of the CVMD Program.
“We’re now aiming to conduct preclinical studies to determine if dietary LPC omega-3 can help to re-myelinate damaged axons in the brain,” added Prof Silver. “Our hope is that supplements containing these fats can help to maintain—or even improve—brain myelination and cognitive function during aging.”
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